genetics
Last reviewed 01/2018
- myophosphorylase normally initiates glycogen breakdown by removing 1,4-glucosyl
groups from the glycogen molecule with release of glucose-1-phosphate
- the majority patients with McAd have undetectable myophosphorylase activity and, thus, are unable to release glucose from glycogen in muscle
- gene for myophosphorylase has been to chromosome 11q13
- family history is positive in about half of the patients and consistent with autosomal recessive inheritance. However there does appear to be an autosomal dominant transmission in some families
Reference:
- Bollig G et al. McArdle's disease and anaesthesia: Case reports. Review of potential problems and association with malignant hyperthermia. Acta Anaesthesiologica Scandinavica 2005;49 (8): 1077-1083.